Fig. 5From: GPR109A controls neutrophil extracellular traps formation and improve early sepsis by regulating ROS/PAD4/Cit-H3 signal axisGPR109A knockout reduces the sensitivity of NETs formation. Neutrophils from WT mice and GPR109A−/− mice were isolated and stimulated with S. aureus, E. coli and LPS, respectively. The results showed that knockout of GPR109A significantly reduced the sensitivity of NETs formationBack to article page