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Fig. 1 | Experimental Hematology & Oncology

Fig. 1

From: YTHDF1 promotes hepatocellular carcinoma progression via activating PI3K/AKT/mTOR signaling pathway and inducing epithelial-mesenchymal transition

Fig. 1

Bioinformatics analysis indicated that YTHDF1 was high-expressed in HCC and was correlated with poor survival. A GEPIA and B UALCAN two databases were used to analyze the mRNA level of YTHDF1 in HCC samples and normal liver samples. C LinkedOmics and D UALCAN two databases were used to analyze the mRNA level of YTHDF1 in different stages of HCC. E The mRNA level of YTHDF1 in different grades of HCC was analyzed in UALCAN database. F LinkedOmics, G GEPIA, and H Kaplan–Meier Plotter databases were used for survival analysis to evaluate the influence of YTHDF1 on the overall survival of HCC patients. *P < 0.05, **P < 0.01, ****P < 0.0001

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Experimental Hematology & Oncology

ISSN: 2162-3619

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