Fig. 11From: GPR109A controls neutrophil extracellular traps formation and improve early sepsis by regulating ROS/PAD4/Cit-H3 signal axisEffects of NETs and GPR109A on mortality of CLP model mice. CLP models were constructed in WT mice, WT + DNaseI mice and GPR109A−/− mice, with 33 mice in each group. DNaseI (100 µL) was injected at a concentration of 100 μg/mL every 2 days. a, b Mortality of CLP model miceBack to article page